Is a revolutionary cancer drug any better than the natural therapy we already have?

The Cancer revolution is now

The UK press sometimes gets a little slaphappy.

Recently, when the University of Bradford unveiled promising cancer research at the annual British Science Festival, the local media outlets fell all over each another with wild headlines announcing the breakthrough.

My pick for the Way Over the Top Prize goes to the Daily Express with this one: “Smart bomb drug can wipe out cancer.”


But even if this drug really DOES prove to be effective, it won’t hit the market for several years.

Until then, you can make use of the revolutionary anti-cancer smart bomb we’ve already got. But it’s not a drug, it’s not expensive, and the side effects are mild. (Guess that doesn’t make for an exciting enough headline…)

How to fight cancer NOW

Like most drugs, the Bradford smart bomb comes from nature.

A flower called autumn crocus contains a toxic chemical called colchicine that kills human body tissues. But the Bradford team had a bright idea. They synthesized an inactive form of colchicine that can be brought back to full toxicity when it senses the presence of an enzyme produced by solid tumors.

But here’s the key point: When the colchicine regains toxicity, it doesn’t harm healthy cells, but it does attack the enzyme that woke it up, so to speak. And it just so happens that tumors desperately need that particular enzyme to develop the tiny blood vessels that deliver oxygen and nutrients the tumor requires to survive.

If all this really works, the guys from Bradford can go ahead right now and make room on their mantle for their Nobel Prize. But as they say in the cancer research game, “That’s a mighty big if.”

So far, it seems to work in mice. The Bradford team implanted mice with human cancer cells. Over the 60-day trial, the treatment shrunk tumors in every single mouse and completely erased tumors in half the mice. All of this occurred with no visible side effects.

Laurence Patterson, Bradford’s lead researcher, told The Guardian, “Any treatment that is a poison that can be retained and is only active in the tumor is clearly very attractive.”

In addition, the treatment also seeks out tumors that have spread through the body.

That really is impressive. In fact it reminds me of another mouse study I told you about three years ago.

National Institutes of Health researchers implanted mice with three types of aggressive cancer cells: brain, pancreatic, and ovarian. In mice treated with intravenous ascorbic acid (IAA), cancer cell growth was cut in half compared to mice who weren’t treated. And like the colchicine drug, IAA didn’t harm healthy cells.

Unlike the Bradford study, however, the NIH study is by no means the first trial to show that mega doses of vitamin C kill cancer cells while leaving healthy cells intact. And many IAA case studies (which you can read about here) have shown the treatment to be effective against several common types of cancer.

So why does the Bradford study get all the splashy headlines while IAA breakthroughs remain mostly unknown to the general public?

It’s pretty simple. The Bradford team is developing a drug. If human studies show that it really is effective, the researchers, the university, and some lucky drug company stand to make billions of dollars from the patent for the treatment.

But you can’t patent vitamin C. That means it will never be an insanely lucrative cash cow, so it will never make headlines.

It’s the sad truth, but there it is.

The upside is that you can get IAA treatment today. And the best place to get started is on the HSI website.

Click here to go to our Find a Doc feature that lists alternative medicine practitioners throughout the U.S. Some offer IAA therapy and some don’t, but if you find a doctor located near you who doesn’t administer IAA, he can probably refer you to someone in your area who does.

“Smart bomb drug can wipe out cancer” Daily Express, 9/12/11,

“Chemotherapy breakthrough could dramatically reduce side-effects” Alok Jha, The Guardian, 9/12/11,

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