Get a “Skinny Liver” in 12 Weeks With This Beet Compound

What if all of your health problems are being caused by a disease you’ve never been diagnosed with?

If you’re over 60 years old, there’s a 40% chance you have something known as “non-alcoholic fatty liver disease” – or NAFLD.

Inside your body, fat is packing into your liver cells like sludge in a clogged pipe.

And when your liver gets clogged, everything breaks down…

Blood sugar spikes. Energy crashes. Belly fat piles on no matter what you eat.

And your risk of diabetes, heart disease, and liver failure skyrockets.

Most doctors don’t test for it… or even diagnose it. But NAFLD may be sabotaging your health right now.

But here’s the good news… scientists have discovered a powerful beet compound that attacks NAFLD at the source.

And it can start turning that fatty liver skinny in as little as 12 weeks…

The compound is betaine—also called trimethylglycine or TMG.

You’ll find it naturally in beets, spinach, and whole grains. But here’s the problem: you’d need to eat 10 POUNDS of beets daily to get a therapeutic dose!

Betaine is what’s called a methyl donor. That means it donates methyl groups to biochemical reactions happening inside your cells—including the ones that export fat OUT of your liver.

When your liver gets fatty, it’s because fat metabolism has stalled. Fat comes in, but it can’t get back out. It just sits there, accumulating.

Betaine restarts that export process.

Researchers at Duke University ran a 12-month pilot study with patients diagnosed with NAFLD. Participants took betaine daily.

By the end of the trial, liver enzyme levels told the story…

ALT—a marker of liver damage—dropped 39%…

AST—another liver enzyme that signals inflammation—fell 38%…

Those aren’t minor improvements. Those are the kinds of reductions that indicate actual REVERSAL of liver damage.

But researchers wanted to know: does betaine actually reduce the fat itself, or just the inflammation?

A 2025 study tested three different doses—8 grams, 4 grams, and 2 grams daily—over 12 to 24 weeks.

All three doses significantly reduced liver fat accumulation starting at 12 weeks. All three brought liver enzymes back into normal range.

Even the lowest dose worked!

A 2024 study identified the exact pathway: betaine activates something called the BHMT/FTO pathway, which directly improves how your liver handles insulin and processes fat.

When insulin signaling improves, your liver stops hoarding fat and starts burning it.

Betaine supplements are widely available as betaine anhydrous or trimethylglycine (TMG).

The studies used doses ranging from 1.5 to 3 grams daily, taken with meals. Some trials went as high as 6-8 grams with no adverse effects.

Most people start at 1.5-2 grams daily and increase if needed.

To giving you the skinny liver you deserve,

Ray Thatcher
Research Director, Health Sciences Institute

Sources:

• Abdelmalek MF, Angulo P, Jorgensen RA, Sylvestre PB, Lindor KD. Betaine, a promising new agent for patients with nonalcoholic steatohepatitis: results of a pilot study. American Journal of Gastroenterology. 2001;96(9):2711-2717. https://pubmed.ncbi.nlm.nih.gov/11569700/
• Miglio F, Rovati LC, Santoro A, Setnikar I. Efficacy and safety of oral betaine glucuronate in non-alcoholic steatohepatitis. Arzneimittelforschung. 2000;50(8):722-727. https://pubmed.ncbi.nlm.nih.gov/10994156/
• Kathirvel E, Morgan K, Nandgiri G, et al. Betaine improves nonalcoholic fatty liver and associated hepatic insulin resistance: a potential mechanism for hepatoprotection by betaine. American Journal of Physiology-Gastrointestinal and Liver Physiology. 2010;299(5):G1068-G1077. https://pmc.ncbi.nlm.nih.gov/articles/PMC2993168/
• Jiang Y, Pan Y, Rhea PR, et al. A sucrose-enriched diet promotes tumorigenesis in mammary gland in part through the 12-lipoxygenase pathway. Cancer Research. 2016;76(1):24-29. https://pubmed.ncbi.nlm.nih.gov/39154792/
• American Academy of Family Physicians. Nonalcoholic Fatty Liver Disease. American Family Physician. 2006;73(11):1961-1968. https://www.aafp.org/pubs/afp/issues/2006/0601/p1961.html